منابع مشابه
[Structural characterization of the somatostatin receptors on rat cerebrocortical membranes].
We characterized structurally the receptors for somatostatin in rat cerebral cortex by affinity labeling with [125I-Tyr1] somatostatin. [125I-Tyr1] somatostatin was cross-linked to cerebrocortical membranes using photoreactive cross-linker: N-5-azido-2-nitrobenzoyloxy-succinimide. Analysis by autoradiography revealed a broad band centered at Mr = 72,000 in the presence or absence of dithiothrei...
متن کاملUptake of opioid drugs by rat cerebrocortical brain slices.
3. H. Okuda, A, Hiratsuka, H. Nojima and T. Watabe, Biochem. Pharmac, 35. 535 (1986). 4. B. N. Ames. J, McCann and E. Yamasaki, Murat. Res. 31. 347 (1975), 5. T. Watabe, T. Ishizuka, N. Ozawa and M. Isobe, Biochem. Pharmac. 31, 2542 (1982). 6. C. C. Irving and R. A. Veazey, Biochim. biophys. Acta 166,246 (1968). 7. T. Watabe, T. lshizuka, T. Fujicda, A. Hiratsuka and K. Ogura, Jpn, J. Cancer Re...
متن کاملMitochondrial and extramitochondrial apoptotic signaling pathways in cerebrocortical neurons.
In cultured cerebrocortical neurons, mild excitotoxic insults or staurosporine result in apoptosis. We show here that N-methyl-d-aspartate (NMDA) receptor-mediated, but not staurosporine-mediated, apoptosis is preceded by depolarization of the mitochondrial membrane potential (Deltapsi(m)) and ATP loss. Both insults, however, release cytochrome c (Cyt c) into the cytoplasm. What prompts mitocho...
متن کاملCerebrocortical Beta Activity in Overweight Humans Responds to Insulin Detemir
BACKGROUND Insulin stimulates cerebrocortical beta and theta activity in lean humans. This effect is reduced in obese individuals indicating cerebrocortical insulin resistance. In the present study we tested whether insulin detemir is a suitable tool to restore the cerebral insulin response in overweight humans. This approach is based on studies in mice where we could recently demonstrate incre...
متن کاملPalmitoylethanolamide Inhibits Glutamate Release in Rat Cerebrocortical Nerve Terminals
The effect of palmitoylethanolamide (PEA), an endogenous fatty acid amide displaying neuroprotective actions, on glutamate release from rat cerebrocortical nerve terminals (synaptosomes) was investigated. PEA inhibited the Ca²⁺-dependent release of glutamate, which was triggered by exposing synaptosomes to the potassium channel blocker 4-aminopyridine. This release inhibition was concentration ...
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ژورنال
عنوان ژورنال: Ankara Üniversitesi Veteriner Fakültesi Dergisi
سال: 1988
ISSN: 1300-0861
DOI: 10.1501/vetfak_0000001115